Micronutrient deficiencies affect more than two billion people of all ages in both developing and industrialized countries. Micronutrient deficiency adversely affects the physiology and immunology of an individual, causing long term health consequences.[2] They are the cause of some diseases, exacerbate others and are recognized as having an important impact on worldwide health. Important micronutrients include iodine, iron, zinc, calcium, selenium, fluorine, and vitamins A, B6, B12, B1, B2, B3, B9 and C.[3]
The population mostly affected by micronutrient deficiency are infants, children and adolescents, women of childbearing age, pregnant women and the elderly.[4] Micronutrient deficiencies are associated with 10% of all children's deaths,[5] and are therefore of special concern to those involved with child welfare. Early childhood micronutrient deficiency leads to stunted growth and impaired cognitive development.[6] Deficiencies of essential vitamins or minerals such as Vitamin A, iron, and zinc may be caused by long-term shortages of nutritious food or by infections such as intestinal worms.[7] They may also be caused or exacerbated when illnesses (such as diarrhoea or malaria) cause rapid loss of nutrients through feces or vomit.[8] There are several interventions to improve the micronutrient status including fortification of foods, supplementation and treatment of underlying infections.[9] Implementation of appropriate micronutrient interventions has several benefits, including improved cognitive development, increased child survival, and reduced prevalence of low birth weight.[10]
A number of elements are known to be needed in these small amounts for proper plant growth and development.[12] Nutrient deficiencies in these areas can adversely affect plant growth and development. Some of the best known trace mineral deficiencies include: zinc deficiency, boron deficiency, iron deficiency, and manganese deficiency.
Boron is believed to be involved in carbohydrate transport in plants; it also assists in metabolic regulation. Boron deficiency will often result in bud dieback.
Copper is a component of some enzymes and of vitamin A. Symptoms of copper deficiency include browning of leaf tips and chlorosis.
Iron is essential for chlorophyll synthesis, which is why an iron deficiency results in chlorosis.
Manganese activates some important enzymes involved in chlorophyll formation. Manganese deficient plants will develop chlorosis between the veins of its leaves. The availability of manganese is partially dependent on soil pH.
Molybdenum is essential to plant health. Molybdenum is used by plants to reduce nitrates into usable forms. Some plants use it for nitrogen fixation, thus it may need to be added to some soils before seeding legumes.
↑Campos Ponce, M., Polman, K., Roos, N., Wieringa, F., Berger, J. and Doak, C., 2018. What Approaches are Most Effective at Addressing Micronutrient Deficiency in Children 0–5 Years? A Review of Systematic Reviews. Maternal and Child Health Journal, 23(S1), pp.4-17.
↑Hwalla, N., Al Dhaheri, A., Radwan, H., Alfawaz, H., Fouda, M., Al‐Daghri, N., Zaghloul, S. and Blumberg, J., 2017. The Prevalence of Micronutrient Deficiencies and Inadequacies in the Middle East and Approaches to Interventions. Nutrients, 9(3), p.229.
↑Adair, L. S., Fall, C. H., Osmond, C., Stein, A. D., Martorell, R., Ramirez-Zea, M., et al. (2013). Associations of linear growth and relative weight gain during early life with adult health and human capital in countries of low and middle income: Findings from five birth cohort studies. Lancet, 382, 525–534.
↑Campos Ponce, M., Polman, K., Roos, N., Wieringa, F., Berger, J. and Doak, C., 2018. What Approaches are Most Effective at Addressing Micronutrient Deficiency in Children 0–5 Years? A Review of Systematic Reviews. Maternal and Child Health Journal, 23(S1), pp.4-17.
↑Bhutta, Z. A., Das, J. K., Rizvi, A., Gaffey, M. F., Walker, N., Horton, S., et al. (2013). Evidence-based interventions for improvement of maternal and child nutrition: What can be done and at what cost? Lancet, 382, 452–477.